Autosomal dominant cerebellar ataxia type I: A review of the phenotypic and genotypic characteristics

Table 2 Differential diagnosis of cerebellar ataxia other than ADCA

Drug Effect	Phenytoin, 5-fluorouracil, cytosine arabinoside, bismuth (Pepto-Bismol^®), mercury-containing fungicides, and lithium
Toxin	Ethanol, methyl mercurial compounds, solvents containing toluene and metals such as lead, manganese, and tin
Nutritional	Vitamin E deficiency, thiamine deficiency (Wernike-Korsakoff syndrome), Gluten sensitivity (Celiac sprue)
Endocrinopathy	Hypothyroidism and hypopituitarism
Infection	HIV, varicella, Epstein-Barr, prion (Creutzfeldt-Jakob, Kuru etc.)
Postinfection	Guillain-Barre and Bickerstaff's encephalitis
Structural or lesional	Ischemic infarction, hemorrhage, neoplasm, demyelination, abscess etc.
Neurodegeneration	Multiple systems atrophy and recessively inherited cerebellar ataxias
Paraneoplastic	Small cell, breast, and ovarian cancer
Other	Idiopathic late onset cerebellar ataxia and ataxia with antiglutamate decarboxylase antibodies