Fig. 4From: A recurrent ABCC2 p.G693R mutation resulting in loss of function of MRP2 and hyperbilirubinemia in Dubin-Johnson syndrome in ChinaMRP2 p.G693R mutant exhibited decreased organic anion transport activity. Compared with wild-type MRP2, the MRP2 p.G693R mutant exhibited decreased organic anion transport activity in HEK293A, Huh-7 and HepG2 cell lines. The Student’s test was used to evaluate the differences of organic anion transport activity. *P < 0.05Back to article page